pancreatic beta cells การใช้

• K v 3.2 is also expressed in pancreatic beta cells.
• Many drugs impair insulin secretion and some toxins damage pancreatic beta cells.
• Calcineurin along with NFAT, may improve the function of diabetics'pancreatic beta cells.
• To compensate for the increased blood glucose levels, fetal pancreatic beta cells can undergo hyperplasia.
• Liraglutide leads to insulin release in pancreatic beta cells in the presence of elevated blood glucose.
• Then, there is the second component of Type 2 diabetes : those sluggish pancreatic beta cells.
• Although pancreatic beta cells produce adequate insulin in infancy, the capacity for insulin production declines thereafter.
• In addition, the protein stimulated insulin secretion from the pancreatic beta cells of both rats and mice.
• Sox4 facilitates differentiation of lymphocytes, osteoblasts, pancreatic beta cells and along with Sox11 promotes neural differentiation.
• Pancreatic beta cells are responsible for making insulin; decreased beta cell activity is associated with DM2 in adulthood.
• In type 1, pancreatic beta cells in the islets of Langerhans are destroyed, decreasing endogenous insulin production.
• In pancreatic beta cells, ATP is the primary metabolic source, and the ATP / release of insulin.
• In pancreatic beta cells FOXO1 mediates glucagon-like peptide-1 effects on pancreatic beta-cell mass.
• CTGF is also important for pancreatic beta cell development, and is critical for normal ovarian follicle development and ovulation.
• These receptors are highly expressed on pancreatic beta cells and are critical regulators of glucose homoestasis by modulating insulin secretion.
• Sulfonylureas are insulin secretagogues, triggering insulin release by direct action on the K ATP channel of the pancreatic beta cells.
• As IGF2 promotes development of fetal pancreatic beta cells, it is believed to be related to some forms of diabetes mellitus.
• Last year, Johnson testified before Congress in favor of research to clone pancreatic beta cells to provide a cure that insulin cannot.
• It thus functions as a synergistic partner to insulin, with which it is cosecreted from pancreatic beta cells in response to meals.
• It also increased insulin sensitivity by about 50 %, improved insulin-stimulated glucose uptake, and protect pancreatic beta cell function.